Tetracycline Drug Uses

Use Tetracycline to treat or prevent infections that are proven or strongly suspected to be caused by susceptible bacteria.

How Taken

The usual daily dose is 1g to 2g. Your doctor may increase the dosage in case of severe infections. You should continue therapy for at least 24 to 48 hours after the symptoms and fever have subsided.

Tetracycline Warnings/Precautions

Talk to your physician before taking this medicine if you are hypersensitive to tetracyclines. Using Tetracycline in the absence of a proven or strongly suspected bacterial infection or a prophylactic indication is unlikely to provide benefit and increases the risk of the development of drug-resistant bacteria.

Tetracycline Missed Dose

If you skip doses or do not complete the full course of therapy, you may risk a decrease in the effectiveness of the immediate treatment. Also there is a chance that bacteria will develop resistance and will not be treatable by Tetracycline or other antibacterial drugs in the future.

Tetracycline Possible Side Effects

Side effects you may experience may include: anorexia, epigastric distress, nausea, vomiting, diarrhea, bulky loose stools, stomatitis, sore throat, glossitis, black hairy tongue, dysphagia, hoarseness, enterocolitis, and inflammatory lesions (with candidal overgrowth) in the anogenital region.

Tetracycline Storage

Store the tablets at room temperature; avoid excessive heat. Dispense in tight, light-resistant containers. Keep out of the reach of children.

Tetracycline Overdose

In case of overdosage, seek emergency medical attention.

More Information

If you are undergoing long-term therapy, periodic laboratory evaluation of organ system function, including renal, hepatic, and hematopoietic systems, should be performed.

Disclaimer

This drug information is for your information purposes only, it is not intended that this information covers all uses, directions, drug interactions, precautions, or adverse effects of your medication. This is only general information, and should not be relied on for any purpose. It should not be construed as containing specific instructions for any particular patient. We disclaim all responsibility for the accuracy and reliability of this information, and/or any consequences arising from the use of this information, including damage or adverse consequences to persons or property, however such damages or consequences arise. No warranty, either expressed or implied, is made in regards to this information.

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Hospitals have germs. And germs have a remarkable ability to develop resistance to the antibiotics we rely on to kill them. These facts are well known to the medical community and have been a source of great concern for years. A number of books and articles for both the lay reader and the professional have been written on the subject, each in its way raising a red flag that says, in one way on another, we have to control the use of antibiotics less we lose them to smart bacteria that learn to become immune to their toxic effects.
Michael Shnayerson and Mark J. Plotkin, authors of The Killers Within: The Deadly Rise of Drug-Resistant Bacteria, have now joined the army of alert writers who are concerned about the dangers of antibiotic resistance and its implications for the return of infectious diseases that cannot be effectively treated. The authors argue that today’s hospitals are not only spreading bacteria from patient to patient, but are also harboring a tougher breed of bug—one that is resistant to antibiotics.
Most infections are either bacterial or viral, and bacterial infections are susceptible to antibiotics. These drugs are designed to attach to enzymes on bacterial cell walls, either preventing the microbes from replicating or killing them outright. Unless, that is, the bacteria mutate and change their enzymes, thus preventing the drug from attaching.
Penicillin was greeted as a panacea when it was developed in the early-20th century. In the decades since, the authors claim—as have others before them—that the overuse of antibiotics for every possible illness has “educated” bacteria, creating opportunities for mutations to occur: “If misuse of antibiotics created drug resistance in the first place, poor infection control in hospitals allowed the bugs to spread.”
Bacteria may be small, but these one-celled organisms can divide and reproduce into more daughter cells than the human population of Earth in just fourteen hours.
Plotkin, an ethnobiologist, and Shnayerson, a contributing editor to Vanity Fair, report what they have learned from interviews and the scientific literature about how bacteria develop resistance and the role genes play in this process. They express admiration for bacteria’s clever defense mechanisms, including the evolution of enzymes that attack antibiotics and tiny pumps that vomit the drug out of the cell.

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